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Re: Viruses and Chimera



Hi John,
See interposed.
Regards,
Tim
 
-----Original Message-----
From: ROSEN Forum [mailto:***On Behalf Of John M
Sent: Tuesday, September 30, 2003 5:58 PM
To: ***
Subject: Re: Viruses and Chimera

Tim,
 
it all depends how does one define "live". Then again in your consideration(s) the ubiquitous reductionistic-scientific narrow 'model' definition also fights your more lax Rosennean complexity view: a sure chance of a clash.
Just for the fun of it:
1.
>"a virus is not alive insofar as an entity unto itself it cannot 1) reproduce, 2) is not a true cell, 3) it has no metabolism or internal processes."<
Are you alive? You, as an entity, cannot reproduce,  you may have true cells among other stuff, but "are" not one,  
 
These 3 points are what I referred to as being the "common" or usual thinking about viruses, and I said later that I thought that the debate was incorrect. Part of that incorrectness is due to incorrect criteria, especially #1 and #2 above.
 
 and I would not exclude a virus's internal processes by timing: that such do occur only after the combination with other cells.  Of course then you call it a chimera. In the parlance the 'virus' causes the pneumonia.
Is a sperm a virus? or the "real cell" egg, for that matter? Is a fetus a parasite?
 
I am not concerned in my post about the designation "parasite". (I have read that viruses are categorized as "obligatory intracellular parasites".) I think that the pejorative term "parasite" is worthy of a post of its own.
Its funny you mention "sperm as virus" - I was wondering about that. I doubt that sperm meet the typical definition of virus. Functionally, though, there seems to be some interesting similarities.
 
 
 
 2.
>"A virus on its own has no volition, no internal metabolic processes at all. It is inert. It does not "seek" a host cell; instead, it randomly floats around until it is in proximity to one. Once in proximity, if the virus and cell are of the right types, the virus will become attached (chemically bind?) to a receptor site on the cell."<
If a virus "gets attached" it was not inert. Your 'volition' includes chemical affinity? in that case an acetic acid molecule has volition?  
 
As far as I know, a virus does not reactively or proactively adjust itself in order to attach. So there is no volition, just chemical affinity.
 
 And the internal (metabolic and other) processes function, once activated, just as in any other living organism which all require activation by enzymes, stimuli, heat, etc. for functioning. By 'activation' through the host a virus reproduces and the transfer of DNA-RNA (and who knows what else) may mean a (special) metabolic activity.
3.
I did not want to use the word: it anticipates the symbiosis with a host (or rather a parasitic cohabitation?) I wonder where to draw the line between such and a chimera-formation (generally speaking, not as one example).  
 
I wondered about the same thing. However, there is (as far as I know) no closed causal loops at this level of organization in a virus that would be a typical prerequisite of an anticipatory system.
 
 Also: where to draw the line between sickness-causing viruses and other (viruslike functional) additives which are necessary in our life-process? 
 
I'm not sure about this. I don't know enough about this aspect of biology to know what "viruslike functional additives" are.
 
4.
I don't quote, but there is a long line of arguing how the parts have not identical functions to the assemblage (complexity). In the eucaryote-evolution the "live" cell penetrating the other "live cell" as forming later the nucleus is 'not live anymore', just part of the ensemble tghe live eucaryotic cell, which is not identical anymore with "the other" procaryote, the citoplasmic originator, or the mitochondrium-originator. The virus(part) of the (infected?) cell is living within your chimera. The chimera is live. 
 
Yes, that evolutionary perspective (and Margulis' symbiogenesis) helped give me the idea of the virus+cell as chimera. 
 
I suggest to look at these things with a more 'wholistic' eye and pay less attention to debatable details drawn upon narrowly cut models of the reductionist science.  
 
To me, an area of controversy in reductionist science is very likely to be symptomatic of an important weakness in the comprehensiveness of that reductionist framework. So I consider such areas to be fertile ground for potentially reframing the controversy in a broader, more comprehensive framework (in this case, the Rosennean framework) that allows coherent resolution of the controversy. 
 
 Rosen could not write a book saying: Hey, Fellows, you got it all wrong... he had to abide by the language and terms of the profanum vulgus of scientists, his readers in spe.  
 
But at points where their "profanum vulgus" failed to be adequate, Rosen introduced new terminology.  Othewise, he could not have made many of his arguments. Of course, as you say, he does "speak a different language" because of that, and that makes some of his points hard to comprehend.
 
 Even in his explanations to Judith (Sorry, Judith, there is no put-down in this) he could not expect his daughter to be at the 'complexiologistic' thinking level what he attained in his many years of studying. He had to use understandable and believable metaphors if he wanted to get appreciation and understanding.
Otherwise maybe even Judith would have thought that he speaks a "different" language.
 
Tim, your post is interesting and well written, I think there are more stringent topics to think of. I would not start a debate with the (reductions science) enemy on their own turf.  
 
Better to tear up their turf, than mine. <g>
 
At any rate I would not penetrate a field, very controversial  in itself, with my new ideas 
controversial enough without the added excitement of a debated situation in itself.
 
My negative point may (at least partially) come from my scanty education in biology.
 
Best regards
 
John M
 
---- Original Message -----
From: Tim Gwinn
To: ***
Sent: Tuesday, September 30, 2003 12:33 PM
Subject: Viruses and Chimera

To all,
 
Since the recent discussion regarding the definition of life, I have been thinking some about this topic. I realized that one useful area to look at might be the contentiousness in biology surrounding whether or not a virus is "alive". 
 
I had some offline conversations with Judith on this topic, and she disagrees with some of my points. It seemed to both of us like an interesting topic for list discussion.
 
After reading some webpages, it seems to me that the common thinking goes roughly this way: a virus is not alive insofar as an entity unto itself it cannot 1) reproduce, 2) is not a true cell, 3) it has no metabolism or internal processes. And the argument on the other side is that once a virus "takes over" a host cell, then it "controls" that cell and uses the machinery of the cell to perform processes and reproduce, so in this (extended) sense the virus is alive.
 
It strikes me, though, that this debate is all wrong.
 
A virus on its own has no volition, no internal metabolic processes at all. It is inert. It does not "seek" a host cell; instead, it randomly floats around until it is in proximity to one. Once in proximity, if the virus and cell are of the right types, the virus will become attached (chemically bind?) to a receptor site on the cell. In response, the virus then apparently initiates a fixed routine to inject its DNA/RNA into the cell's cytoplasm.
 
To speak of this sequence in active volitional language, such as "the virus seeks out a host cell, uses its DNA/RNA to take control of the host cell", seems to me entirely inappropriate, and assigns volitional capabilities to a structure that has no such capabilities.
 
I would argue that, on this level of description, a virus is a Rosennean mechanism, not a complex system. The virus on its own has no internal processes, much less any internal causal closed loops of processes. Its one process of injecting DNA/RNA into a cell is in reaction to the successful binding to the host. Crudely, it is like a spring-loaded clamp and hypodermic attached to a balloon. It floats around randomly until it comes in contact with something (say, some organism) that trips the spring clamp, which then causes the hypodermic plunger to move. This is an entirely fixed and mechanical sequence.
 
Also, regarding viruses as "evolving" entities. I think the idea that the "evolution" of viruses is evidence that viruses are alive somehow is very weak. Prions (so-called "misfolded" proteins) replicate and apparently also evolve (which may be what allows them to sometimes pass between species), but no one (to my knowledge) suggests that this indicates that prions are alive.
 
So, the notion that once the virus is attached to the cell, that the virus "takes the cell over" is erroneous. Just as it would be erroneous to say that the balloon-clamp-hypodermic mechanism "takes over" an organism it attaches to and injects with something that alters the processes in the organism. Similarly, it would be erroneous to conclude that the balloon-clamp-hypodermic is therefore now properly called "alive" because it has attached itself to a living organism and has altered what the organism does.
 
 
Instead, I think the correct view is that the combining of the virus and the host cell form a chimeric relationship: the combination becomes a new creature. This new creature has, as one of its functions, to generate new copies of the virus. Depending on the type of virus, this creature may then dissolve, turn cancerous, or continue to persist.
 
Rosen talked about hermit crabs in ch. 18 of Essays and the way in which the crab adopts a snail shell and collects sea anemones and becomes a new chimeric organism with a new set of behaviors. It seems to me that this is exactly the type of case that is occurring in the virus-host situation. However, I think this has been historically misinterpreted perhaps because of the apparent similarity of the symptoms arising from viral infections to bacterial ones, and probably moreso because a virus has RNA/DNA, which is taken as putative evidence of life. So, the virus has been incorrectly seen as purposively usurping control of the host, leading to the supposition that a virus is alive somehow.
 
But the virus by itself is much like the snail shell by itself: it is inert and not alive. When it has become combined with the crab, one cannot ask: is the shell now alive or not by virtue of this combining? The question is improper, since the shell is not now a thing by itself, but is now a part of this larger organism, the chimera, and that chimera is what is alive. Similarly, the virus does not become alive by virtue of combining with the host cell; instead, both are subsumed into this new larger chimera organism.
 
I hate to use the old saw, but a chimera is a case where one must adhere to the principle "the whole is greater than the parts". Once combined, the virus and cell cannot continue to be thought of as distinct entities or parts if we want to explain and understand the behaviors of the result of this combining. Otherwise, I think this only leads to the confused thinking that perpetuates the virus live-or-dead debate.
 
The tendency toward reductionist thinking, and of thinking of the virus as a volitional creature, leads to one attempting to mentally maintain the virus and host as distinct and separable entities, even after they have combined. The generation or replication of copies of the initial virus also seems to support this view. But I think that this is simply trying to make the situation appear other than it actually is, which is that the (virus + host) once joined are a new creature with new functionality (just as the (hermit crab + shell + anemone) is a new chimeric creature). One of those functions is to process raw materials into copies of the original virus. And the question of "what is alive?" at that point is answered by: "this new creature, the chimera, is what is alive".
 
 
Regards,
Tim